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Naxocina (Azithromycin)

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Azalides are a class of macrolide antibiotics which contain a nitrogen in the macrolide ring. This imparts different pharmacokinetic properties and is associated with greater stability of the molecule. One such Azalide is the antibiotic Naxocina. This medication is a macrolide antibiotic used for various bacterial infections such as infections of the middle ear, throat, bronchus, sinuses, skin and soft tissue. It is also useful in treating pneumonia, typhoid, gonorrhoea, granuloma inguinale and chancroid. It prevents bacterial growth.

Other names for this medication:
Amovin, Azalid, Azibiot, Azicin, Azicine, Azicip, Azimax, Azimycin, Azithral, Azithro, Azitrom, Azitromicina, Azitrox, Aziwok, Azomac, Azomex, Bezanin, Doyle, Imbys, Macromax, Magnabiotic, Mezatrin, Neozith, Nifostin, Novozithron, Ordipha, Phagocin, Respazit, Rozith, Simpli, Sitrox, Tanezox, Texis, Tremac, Tritab, Trozocina, Ultrabac, Zicho, Zirocin, Zistic, Zithrin, Zithrocin, Zitrex, Zymycin

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Chloromycetin, Cipro, Omnicef, Flagyl, Ceftin, Cefixime, Cleocin, Terramycin

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Also known as: Azithromycin


Naxocina is in a group of drugs called macrolide antibiotics. Naxocina fights bacteria in the body. Naxocina is used to treat many different types of infections caused by bacteria, such as respiratory infections, skin infections, ear infections, and sexually transmitted diseases. Naxocina may also be used for purposes other than those listed in this medication guide.

To reduce the development of drug-resistant bacteria and maintain the effectiveness of Naxocina Tablets and other antibacterial drugs, Naxocina Tablets should be used only to treat infections that are proven or strongly suspected to be caused by susceptible bacteria. When culture and susceptibility information are available, they should be considered in selecting or modifying antibacterial therapy. In the absence of such data, local epidemiology and susceptibility patterns may contribute to the empiric selection of therapy.

Naxocina Tablets are a macrolide antibacterial drug indicated for the treatment of patients with mild to moderate infections caused by susceptible strains of the designated microorganisms in the specific conditions listed below.

Naxocina is an antibiotic used to treat bacterial infections of the nose, throat, lungs, bronchitis, ear, skin, soft tissues, and sexually transmitted genital and urinary infections.

Naxocina is a semi-synthetic macrolide antibiotic of the azalide class. Like other macrolide antibiotics, Naxocina inhibits bacterial protein synthesis by binding to the 50S ribosomal subunit of the bacterial 70S ribosome. Binding inhibits peptidyl transferase activity and interferes with amino acid translocation during the process of translation. Its effects may be bacteriostatic or bactericidal depending of the organism and the drug concentration. Its long half life, which enables once daily dosing and shorter administration durations, is a property distinct from other macrolides.


Use Naxocina as directed by your doctor.

Take Naxocina by mouth with or without food. If stomach upset occurs, take with food to reduce stomach irritation.

Do not take an antacid that has aluminum or magnesium in it within 1 hour before or 2 hours after you take Naxocina.

Naxocina works best if it is taken at the same time each day.

To clear up your infection completely, use Naxocina for the full course of treatment. Keep using it even if you feel better in a few days.

Ask your health care provider any questions you may have about how to use Naxocina.


Seek emergency medical attention if you think you have used too much of this medicine. Symptoms of an Naxocina overdose may include nausea, vomiting, diarrhea, and stomach discomfort.


Store medicines at room temperature, away from heat and direct light. Do not freeze medicines unless required by package insert. Keep medicines away from children and pets.

Do not flush medications down the toilet or pour them into drainage unless instructed to do so. Medication discarded in this manner may contaminate the environment. Please consult your pharmacist or doctor for more details on how to safely discard Naxocina Tablet.

Side effects

The most common side effects associated with Naxocina are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


You should not use this medication if you have ever had jaundice or liver problems caused by taking azithromycin. You should not use azithromycin if you are allergic to it or to similar drugs such as erythromycin (E.E.S., EryPed, Ery-Tab, Erythrocin, Pediazole), clarithromycin (Biaxin), telithromycin (Ketek), or troleandomycin (Tao).

There are many other medicines that can interact with azithromycin. Tell your doctor about all medications you use. This includes prescription, over-the-counter, vitamin, and herbal products. Do not start a new medication without telling your doctor. Keep a list of all your medicines and show it to any healthcare provider who treats you.

Take this medicine for the full prescribed length of time. Your symptoms may improve before the infection is completely cleared. Skipping doses may also increase your risk of further infection that is resistant to antibiotics. Azithromycin will not treat a viral infection such as the common cold or flu.

Avoid taking an antacid within 2 hours before or after you take azithromycin. Some antacids can make it harder for your body to absorb azithromycin.

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This study was conducted in all hospitals providing rectal cancer surgery in a Canadian province. We found strong evidence of degeneration across the genus in the nonrecombining regions of the mating-type chromosomes, with significantly higher rates of nonsynonymous substitution (dN/dS) than in nonmating-type chromosomes or in recombining regions of the mating-type chromosomes. Connecting ACF to histones may be prerequisite for efficient conversion of ATP-dependent conformational changes of ISWI into translocation of DNA relative to the histones during nucleosome mobilization. Intracranial aspergillosis producing cavernous sinus syndrome and rupture of internal carotid artery. Variation of standard technics for correction of blepharoptosis. Human infections with avian influenza are rare and this is the first time that human infection with a low pathogenic avian influenza A virus has been associated with fatal outcome. Although the majority of uremic patients are insulin resistant and about half of them are glucose intolerant, they are rarely diabetics.

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These exposures were accompanied by influx of alveolar macrophages, giant cell formation, and a granulomatous response in the lung. The effects of amygdaloid lesions on different measures of aversive learning are discussed. The first two remains date to approximately 39 ka 14C BP ( approximately 43.5 ka cal BP), whereas the latter two derive from earlier in oxygen isotope stage 3. In order to establish early and precise methods for evaluating the effect of secondary chemotherapy in patients with recurrent ovarian cancer, both the clinical course of 3 women treated with gemcitabine-based secondary chemotherapy and the potential for early and accurate evaluation of the secondary chemotherapeutic effect of 18F-fluorothymidine (FLT) PET are reported. Prior incubation in buffered peptone water supplemented with ferrioxamine E noroxin drug class markedly extended the time that bacteria were recoverable by plating, except in the case of mutants deficient in ferrioxamine E uptake.

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Bipolar recordings from the coaxially moveable electrode catheter provide unique electrogram data. Recovery profiles were not statistically different when the fluorescence from iron-free cytochrome c or fluorescein-labeled cytochrome c was used and were essentially the same in the presence or absence of an uncoupler. When the mandibular third molar and the mandibular canal were shown to have direct contact in CBCT images, the prevalence of nerve damage was higher than in other cases. These data suggest that stenting does influence cuspal calcification of AOA-treated, glutaraldehyde-fixed porcine aortic valves. Here, we demonstrate the effects of PSCs and TSCs on tumorigenesis in prostate cancer (PCa) and identify the mechanisms underlying these effects. A pathophysiological link between this mutation and insulin resistance remains unclear. Transforming growth factor alpha (TGF-alpha), TGF-beta and galanin mediate the action of estrogen on the postnatal expansion of the lactotrope cell population and expression of the gene encoding prolactin in an autocrine/paracrine manner. This should be facilitated by appropriate training programmes and the implementation of quality assurance procedures at each stage of the cleaning process.

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