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Claritrox (Clarithromycin)
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Claritrox

Claritrox belongs to a group of medicine called macrolide antibiotics derived from Saccharopolyspora erythraea (originally called Streptomyces erythreus), used to treat a wide range of bacterial infections including chest infections such as bronchitis and pneumonia, skin problems such as cellulitis (potentially serious bacterial skin infection.

Other names for this medication:
Biclar, Binoclar, Clabel, Clamycin, Clarimac, Clarimycin, Claripen, Clarithromycin, Claritron, Clariva, Claromac, Claromycin, Claron, Clarytas, Clasine, Ezumycin, Finasept, Geromycin, Infex, Klabet, Klarifor, Klarimac, Klarith, Klaritpharma, Klazidem, Klonacid, Larithro, Macrol, Mononaxy, Nutabact, Onexid, Rolicytin, Veclam, Vikrol, Xylar

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Also known as: Clarithromycin

Description

Claritrox (generic name: clarithromycin; brand names include: Maclar / Klaricid / Klacid / Clarimac / Claribid) is used to treat many different types of bacterial infections affecting the skin and respiratory system, including: Strep throat, Pneumonia, Sinusitis (inflamed sinuses), Tonsillitis (inflamed tonsils), Acute middle ear infections, Acute flare-ups of chronic bronchitis.

It also is used to treat and prevent disseminated Mycobacterium avium complex (MAC) infection [a type of lung infection that often affects people with human immunodeficiency virus (HIV)]. It is used in combination with other medications to eliminate H. pylori, a bacteria that causes ulcers.

It also is used sometimes to treat other types of infections including Lyme disease (an infection that may develop after a person is bitten by a tick), crypotosporidiosis (an infection that causes diarrhea), cat scratch disease (an infection that may develop after a person is bitten or scratched by a cat), Legionnaires' disease (a type of lung infection), and pertussis (whooping cough; a serious infection that can cause severe coughing). It is also sometimes used to prevent heart infection in patients having dental or other procedures.

This medication may be prescribed for other uses; ask your doctor or pharmacist for more information.

Claritrox works by stopping the growth of or killing sensitive bacteria by interfering with their protein synthesis.

Dosage

Claritrox is available in tablets. Take Claritrox orally. Take Claritrox with full glass of water. Take Claritrox with or without food. Do not crush, chew, or break the Claritrox tablet. Swallow the pill whole. Shake the Claritrox oral suspension well before measuring a dose. Measure the Claritrox oral suspension with a marked measuring spoon or medicine cup. Take Claritrox for for 7 to 14 days. The dosage and the kind of medication depend on the disease and its prescribed treatment. Do not stop taking Claritrox suddenly.

Overdose

Overdose symptoms may include severe stomach pain, nausea, vomiting, or diarrhea.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Protect from light. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Claritrox are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Discontinue immediately if hepatitis or severe hypersensitivity reactions occurs. Severe renal impairment. Proarrhythmic conditions (eg, hypokalemia, hypomagnesemia, bradycardia); avoid. Myasthenia gravis. History of porphyria; avoid concomitant ranitidine bismuth citrate. Elderly. Pregnancy (Cat.C): usually not recommended. Nursing mothers.

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Because PPARgamma ligands exert partially overlapping but distinct subsets of biological action through PPARgamma binding, we wished to examine whether interactions between PPARgamma and known coactivators were induced to the same extent by different classes of PPARgamma ligand. ACE inhibitor and calcium channel blocker in a fixed combination. Long-term treatment with tamoxifen facilitates translocation of estrogen receptor alpha out of the nucleus and enhances its interaction with EGFR in MCF-7 breast cancer cells. Here antibodies generated against a synthetic peptide from the C terminus of human EGF receptor are shown to immunoprecipitate the EGF receptor from human and avian cells, as well as the v-erbB proteins from AEV- zithromax classification antibiotic transformed cells that become phosphorylated on tyrosine residues upon the addition of gamma-32P-ATP. To our knowledge, only one other case of TOSV infection has been reported in this area. Both fractions, however, are the same as judged from sodium dodecylsulfate-polyacrylamide gel electrophoresis. In these cases, first, we apply an artifact removal method and subsequently apply the PARAFAC model on the epilepsy tensor from which potential artifacts have been removed. Here, we report a scheme for focusing light inside scattering media by employing intrinsic dynamics as guide stars.

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Enprostil inhibits post-prandial gastrin release: a dose-response study. The tissue compatibility of the polypropylene mesh material and anterior cervical myocutaneous skin flap makes this a promising therapeutic substitute for treatment of patients with extensive tracheal lesions. Individuals with binge eating disorder (BED) have high rates of comorbid psychopathology, yet little is known about the relation of comorbidity to eating disorder features or response to treatment. Surprisingly, these genes encode proteins with antagonistic activity as two, UBA1 and UBA2, are ubiquitin-activating enzymes whereas the other three are de-ubiquitinating hydrolases. Mucosubstance histochemical study of 33 cholesteatoma tissues was performed to clarify the distribution and character of mucin in the perimatrix. The physical association is independent of cyclin H binding or Cdk7 kinase activity and is conserved between the related Sacharomyces cerevisiae CTD kinase Kin28 and the HIT protein Hnt1. Based on several cases of drug toxicity (or lack thereof) in the breastfeeding infant, individual risk:benefit analyses are discussed. Furthermore, the facilitation was dependent on activation of D1/D5 receptors.

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